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Prostaglandins (PGs) have been implicated as signal molecules, and the changes in their synthesis may be a cause or effect of bone resorption. This study investigated whether 1,25 dihydroxyvitamin D3 could stimulate PGE2 and PGF2 alpha production by osteoblasts. Primary cultures of bovine osteoblasts were grown in alpha-MEM and treated with 1,25 dihydroxyvitamin D3 (10(-9) M). After 24 h, total PGE and PGF were determined in the medium. In controls, PGE2 concentrations were 15.2 +/- 2.1 ng/ml and PGF2 alpha concentrations were 2.6 +/- 0.4 ng/ml. PGE2 concentrations in vitamin D treated cells were significantly increased to 23.8 +/- 3.2 ng/ml, whereas the PGF2 alpha concentration was not changed. Release of both prostaglandins was calcium dependent since calcium deprivation inhibited stimulation of prostaglandin production by vitamin D. Dexamethasone at 10(-7) M also enhanced PGE2 release, but PGF2 alpha production was not affected. PGE2 and PGF2 alpha release induced by 1,25 dihydroxyvitamin D3 may be involved in the stimulation of prostaglandin biosynthesis by bone cells and may provide another mechanism of the dihydroxyvitamin D3 action on bone metabolism./*
* Copyright (c) 2000-2002,2005 Silicon Graphics, Inc.
* All Rights Reserved.
*
* This program is free software; you can redistribute it and/or
* modify it under the terms of the GNU General Public License as
* published by the Free Software Foundation.
*
* This program is distributed in the hope that it would be useful,
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